Recovery time frames are often double or triple the duration of a typical recovery. It’s the same rehab but at a slower pace. You may be frustrated but remember, there’s no magic to recovering from a lesion. They’ll improve with regular ankle sprain rehab so you’ll never know you’ve damaged your cartilage. We only tend to find the worst ones because the smaller or milder injuries often go unnoticed.
About 7%, or 1 in 14, ankle sprains have an associated talar dome lesion. While a defect in joint cartilage may sound and feel nasty, they’re surprisingly common. Why does a talar dome injury take so long to recover? Surgical repair of talar dome lesions are only required for very large defects (typically >1 square cm) as most lesions recover well with non-surgical approaches such as controlled reloading and strength exercises. Crutches usually aren’t required unless it’s a severe case with lots of pain or swelling. You’ll also need to keep the swelling at a minimum using ice packs and compressive bandaging. To improve the early recovery after injury, look at routine supportive taping or bracing to prevent aggravating the cartilage defect. Even if it sees a damaged area, we can’t tell if it’s a new or old lesion (without seeing the reaction). That means a clear X-ray may not mean that you’re clear of injury. Although X-ray may visualise a lesion, it’s hit & miss in actually spotting it and can’t see any bony reaction. The size of this reaction will often affect rehab time frames. MRI will show both the size of the damaged area as well as the reaction in the bone below the injury. Imaging for talar dome lesions is best done with MRI. These lesions are often suspected after 2-4 weeks of unexpectedly slow recovery and ongoing symptoms including fluctuating swelling inside the joint. We just can’t do anything to load that part of the cartilage in isolation to confirm the diagnosis.
#Talar dome professional
The pain is often described as “deep inside the joint”.Ĭlinical testing by a health professional is unable to detect talar dome lesions. It hurts but there’s no spot that you push on to produce the same pain. It’s a consistent dull ache that you can’t put your finger on. It’s called “intracapsular effusion” and it’s a sign that something inside the joint is producing fluid.Ī deep ongoing ache is another sign, although it’s not unique to talar dome lesions. And it’s gone again by the next morning, only to repeat the same pattern. But by mid afternoon, there’s a cherry-sized lump forming on front of your lateral (outside) ankle bone. You wake up just fine, no obvious swelling. Recurrent swelling that tends to fluctuate from morning to evening is another concern. When you rolled it, you thought it’d be 4 weeks to return to running but 8 weeks later, it’s still not recovered enough to try a run. Increased recovery time can be a little vague but it’s basically an ankle sprain that seems to take “forever” to get better. And given that your entire body weight rests on this small cartilage surface, it’s got to deal with some pretty big loads during your daily life.Ĭlassic symptoms include an increased recovery time after an ankle sprain, recurrent swelling with normal activity and an ongoing deep ache during/after activity. Talar dome lesions typically involve damage to the smooth cartilage surface overlying the bone. It’s a sneaky injury that can be a complication of a “typical” ankle sprain because it’s not always obvious in the first few weeks post-injury, even with a clinical assessment. Talar dome lesion refers to an injury to the surface of the bone in the ankle joint, the Talus. If it’s not a talar dome lesion, what else could it be?.Why does a talar dome injury take so long to recover?.Bone marrow edema syndrome: an unusual cause for spontaneous unrelentless bilateral knee pain. doi:10.4081/reumatismo.2014.790Ĭhambel A, Spranger A, Almeida P, do Briot JS. A clinical overview of bone marrow edema, Reumatismo. How we manage bone marrow edema-an interdisciplinary approach. Edema surrounding benign tumors and tumor-like lesions. National Organization for Rare Disorders (NORD). Transient hip osteoporosis: etiopathogenetic, clinical, and imaging approach. Maisi N, Patoulias D, Tsagkaris C, Tsagatakis M, Goules D. Reviewing bone marrow edema in athletes: a difficult diagnostic and clinical approach. Bone marrow edema: pathogenetic features. Molfetta L, Florian A, Saviola G, Frediani B.
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